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TUG-1197.

TUG-1197.

CPL304110 is a potent, orally active and selective inhibitor of fibroblast growth factor receptors FGFR (1-3) , with IC 50 values of 0.75 nM, 0.5 nM, and 3.05 nM for FGFR (1-3), respectivelyIn VitroCPL304110 (0-0.6 μM) dose-dependently inhibits

CPL304110 is a potent, orally active and selective inhibitor of fibroblast growth factor receptors FGFR (1-3) , with IC 50 values of 0.75 nM, 0.5 nM, and 3.05 nM for FGFR (1-3), respectivelyIn VitroCPL304110 (0-0.6 μM) dose-dependently inhibits

CPL304110 is a potent, orally active and selective inhibitor of fibroblast growth factor receptors FGFR (1-3) , with IC 50 values of 0.75 nM, 0.5 nM, and 3.05 nM for FGFR (1-3), respectivelyIn VitroCPL304110 (0-0.6 μM) dose-dependently inhibits

CPL304110 is a potent, orally active and selective inhibitor of fibroblast growth factor receptors FGFR (1-3) , with IC 50 values of 0.75 nM, 0.5 nM, and 3.05 nM for FGFR (1-3), respectivelyIn VitroCPL304110 (0-0.6 μM) dose-dependently inhibits

Potent and selective FFA4 (GPR120) agonist.

InformationTUG-891 is a potent and selective agonist of thelong chain free fatty acid (LCFA) receptor 4 (FFA4/GPR120), which demonstrates both potential opportunity and possible challenges to therapeutic agonism.

InformationTUG-891 is a potent and selective agonist of thelong chain free fatty acid (LCFA) receptor 4 (FFA4/GPR120), which demonstrates both potential opportunity and possible challenges to therapeutic agonism.

InformationTUG-891 is a potent and selective agonist of thelong chain free fatty acid (LCFA) receptor 4 (FFA4/GPR120), which demonstrates both potential opportunity and possible challenges to therapeutic agonism.

InformationTUG-891 is a potent and selective agonist of thelong chain free fatty acid (LCFA) receptor 4 (FFA4/GPR120), which demonstrates both potential opportunity and possible challenges to therapeutic agonism.

TUG-424 is an agonist of the free fatty acid receptor FFA|1|(GPR40) and at 100nM, TUG-424 has been shown to enhance glucose-stimulated insulin secretion in INS-1E cells.