WWL113 is a selective and orally active Ces3 and Ces1f inhibitor, with IC50 values of 120 nM and 100 nM for Ces3 and Ces1f, respectively. WWL113 appears to show excellent selectivity for the 60-kDa serine hydrolase (or hydrolases).
WWL113 is a selective and orally active Ces3 and Ces1f inhibitor, with IC50 values of 120 nM and 100 nM for Ces3 and Ces1f, respectively. WWL113 appears to show excellent selectivity for the 60-kDa serine hydrolase (or hydrolases).
WWL113 is a selective and orally active Ces3 and Ces1f inhibitor, with IC50 values of 120 nM and 100 nM for Ces3 and Ces1f, respectively. WWL113 appears to show excellent selectivity for the 60-kDa serine hydrolase (or hydrolases).
WWL113 is a selective and orally active Ces3 and Ces1f inhibitor, with IC50 values of 120 nM and 100 nM for Ces3 and Ces1f, respectively. WWL113 appears to show excellent selectivity for the 60-kDa serine hydrolase (or hydrolases).
WWL113 is a selective and orally active Ces3 and Ces1f inhibitor, with IC50 values of 120 nM and 100 nM for Ces3 and Ces1f, respectively. WWL113 appears to show excellent selectivity for the 60-kDa serine hydrolase (or hydrolases).
WWL113 is a selective and orally active Ces3 and Ces1f inhibitor, with IC50 values of 120 nM and 100 nM for Ces3 and Ces1f, respectively. WWL113 appears to show excellent selectivity for the 60-kDa serine hydrolase (or hydrolases).
WWL113 is a selective and orally active Ces3 and Ces1f inhibitor, with IC50 values of 120 nM and 100 nM for Ces3 and Ces1f, respectively. WWL113 appears to show excellent selectivity for the 60-kDa serine hydrolase (or hydrolases).
WWL113 is a selective and orally active Ces3 and Ces1f inhibitor, with IC50 values of 120 nM and 100 nM for Ces3 and Ces1f, respectively. WWL113 appears to show excellent selectivity for the 60-kDa serine hydrolase (or hydrolases).
WWL113 is a selective and orally active Ces3 and Ces1f inhibitor, with IC50 values of 120 nM and 100 nM for Ces3 and Ces1f, respectively. WWL113 appears to show excellent selectivity for the 60-kDa serine hydrolase (or hydrolases).
WWL113 is a selective and orally active Ces3 and Ces1f inhibitor, with IC50 values of 120 nM and 100 nM for Ces3 and Ces1f, respectively. WWL113 appears to show excellent selectivity for the 60-kDa serine hydrolase (or hydrolases).
InformationSAR-020106 is an ATP-competitive, potent, and selectiveCHK1inhibitor with an IC50 of 13.3 nM.TargetsChk1 (Cell-free assay) 13.3 nMIn vitroSAR-020106 abrogates an etoposide-induced G2 arrest with an IC50 of 55 nmol/L in HT29 cells, and
InformationSAR-020106 is an ATP-competitive, potent, and selectiveCHK1inhibitor with an IC50 of 13.3 nM.TargetsChk1 (Cell-free assay) 13.3 nMIn vitroSAR-020106 abrogates an etoposide-induced G2 arrest with an IC50 of 55 nmol/L in HT29 cells, and
InformationSAR-020106 is an ATP-competitive, potent, and selectiveCHK1inhibitor with an IC50 of 13.3 nM.TargetsChk1 (Cell-free assay) 13.3 nMIn vitroSAR-020106 abrogates an etoposide-induced G2 arrest with an IC50 of 55 nmol/L in HT29 cells, and
InformationSAR-020106 is an ATP-competitive, potent, and selectiveCHK1inhibitor with an IC50 of 13.3 nM.TargetsChk1 (Cell-free assay) 13.3 nMIn vitroSAR-020106 abrogates an etoposide-induced G2 arrest with an IC50 of 55 nmol/L in HT29 cells, and
InformationSAR-020106 is an ATP-competitive, potent, and selectiveCHK1inhibitor with an IC50 of 13.3 nM.TargetsChk1 (Cell-free assay) 13.3 nMIn vitroSAR-020106 abrogates an etoposide-induced G2 arrest with an IC50 of 55 nmol/L in HT29 cells, and
InformationSAR-020106 is an ATP-competitive, potent, and selectiveCHK1inhibitor with an IC50 of 13.3 nM.TargetsChk1 (Cell-free assay) 13.3 nMIn vitroSAR-020106 abrogates an etoposide-induced G2 arrest with an IC50 of 55 nmol/L in HT29 cells, and
InformationSAR-020106 is an ATP-competitive, potent, and selectiveCHK1inhibitor with an IC50 of 13.3 nM.TargetsChk1 (Cell-free assay) 13.3 nMIn vitroSAR-020106 abrogates an etoposide-induced G2 arrest with an IC50 of 55 nmol/L in HT29 cells, and
InformationSAR-020106 is an ATP-competitive, potent, and selectiveCHK1inhibitor with an IC50 of 13.3 nM.TargetsChk1 (Cell-free assay) 13.3 nMIn vitroSAR-020106 abrogates an etoposide-induced G2 arrest with an IC50 of 55 nmol/L in HT29 cells, and
InformationSAR-020106 is an ATP-competitive, potent, and selectiveCHK1inhibitor with an IC50 of 13.3 nM.TargetsChk1 (Cell-free assay) 13.3 nMIn vitroSAR-020106 abrogates an etoposide-induced G2 arrest with an IC50 of 55 nmol/L in HT29 cells, and
InformationSAR-020106 is an ATP-competitive, potent, and selectiveCHK1inhibitor with an IC50 of 13.3 nM.TargetsChk1 (Cell-free assay) 13.3 nMIn vitroSAR-020106 abrogates an etoposide-induced G2 arrest with an IC50 of 55 nmol/L in HT29 cells, and
InformationSAR-020106 is an ATP-competitive, potent, and selectiveCHK1inhibitor with an IC50 of 13.3 nM.TargetsChk1 (Cell-free assay) 13.3 nMIn vitroSAR-020106 abrogates an etoposide-induced G2 arrest with an IC50 of 55 nmol/L in HT29 cells, and
InformationSAR-020106 is an ATP-competitive, potent, and selectiveCHK1inhibitor with an IC50 of 13.3 nM.TargetsChk1 (Cell-free assay) 13.3 nMIn vitroSAR-020106 abrogates an etoposide-induced G2 arrest with an IC50 of 55 nmol/L in HT29 cells, and