(±)13-HDHA is an autoxidation product of DHA in vitro. It is also produced from incubations of DHA in rat liver, brain, and intestinal microsomes.
(±)13-HDHA is an autoxidation product of DHA in vitro. It is also produced from incubations of DHA in rat liver, brain, and intestinal microsomes.
A fungal metabolite that inhibits cyclooxygenase, blocking both the conversion of AA to PGH2 and the conversion of PGH2 to PGE2 (IC<sub>50</sub>s = 40 and 9 µM, respectively) also inhibits the reverse transcriptase of avian myeloblastosis
A fungal metabolite that inhibits cyclooxygenase, blocking both the conversion of AA to PGH2 and the conversion of PGH2 to PGE2 (IC<sub>50</sub>s = 40 and 9 µM, respectively) also inhibits the reverse transcriptase of avian myeloblastosis
Formed by the cleavage of the sialic acid residue from ganglioside GM1 by neuraminidase a component of lipid rafts serves as a glycolipid receptor for bacterial host response signaling.
Formed by the cleavage of the sialic acid residue from ganglioside GM1 by neuraminidase a component of lipid rafts serves as a glycolipid receptor for bacterial host response signaling.
(±)10-HDHA is an autoxidation product of DHA in vitro. It is also produced from incubations of DHA in rat liver, brain, and intestinal microsomes.
(±)10-HDHA is an autoxidation product of DHA in vitro. It is also produced from incubations of DHA in rat liver, brain, and intestinal microsomes.
A BLT1 receptor antagonist with a Ki value of 159 nM on guinea pig lung membranes does not antagonize the binding of [3H]-LTB4 to the human BLT2 receptor.
A BLT1 receptor antagonist with a Ki value of 159 nM on guinea pig lung membranes does not antagonize the binding of [3H]-LTB4 to the human BLT2 receptor.